By Dr Thomas L Husted, Prof. Dr. Hannes Wacha, Professor Joseph S Solomkin (auth.)
A clinician's guide for intra-abdominal infections written via specialists within the field.
- A guide to assist physicians to fast realize and higher comprehend the pathogenesis of intra-abdominal
- contains a number of top quality black and white and colour photos from real-life scientific cases
- Adheres to an easy layout to function crucial and quickly reference advisor for all non-surgical citizens and physicians
Read or Download Clinician’s Manual on Intra-abdominal Infections PDF
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Additional resources for Clinician’s Manual on Intra-abdominal Infections
5 0'41&$*'*$ %*4&"4&4 t C. difficile causes toxin-mediated colitis, expressed as two exotoxins: toxin A and toxin B. Although toxin A is more pathogenic in vivo, both toxins are responsible for an increase in inflammation, edema, and eventual focal ulceration of the colonic mucosa leading to transmural necrosis. Medical history and physical examination Patients with C. difficile colitis will present with a history of recent (up to the preceding 8 weeks) antibiotic use, abdominal pain, fever, and copious, often uncontrollable, diarrhea.
Post-operative intra-abdominal processes Pathophysiology Peritonitis or abscess presenting in the post-operative period is a complication of concern to most surgeons. Several situations are associated with a higher risk for the development of these complications, including operations performed in emergency settings or in less than ideal circumstances. Similarly, patients with significant comorbid conditions or malnutrition are also at increased risk. Causes include enteral spillage due to disease or during operation, anastomotic leakage due to technical errors, poorly healing tissues, or concurrent infection, which can lead to peritoneal seeding, tissue necrosis, or recurrent infection following surgical and antibiotic treatment (ie, tertiary peritonitis).
5*0/4 A review of 100 appendectomies carried out to find cases of acute appendicitis showed that the patients all presented with signs and symptoms of acute abdominal pain and tenderness in the lower quadrant of the abdomen, and all patients had ultrasound and laboratory test results, including a urine analysis. The intervention was always decided by the senior surgeon. The data showed that only 50% of the laboratory findings correlated with acute appendicitis. The usual characteristics and clinical signs (eg, pain, tenderness of the abdomen) and a thorough and repeated clinical assessment over time were the main factors given by the experienced surgeon to support the need for an operation.